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List Of Drugs That Cause GYNO
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List Of Drugs That Cause GYNO - 01-17-2013, 03:24 PM

– By purplehaze

Drug Side Effects – Gynecomastia Male Breast Enlargement

There are many possible reasons for male breast growth. If caused by a medical problem, this should be corrected first. Such issues can be investigated by your internist or an endocrinologist. In most cases there is no known cause. A complete discussion of the many possible causes for gynecomastia is beyond the scope of this introduction.

These drugs (according to the Physician Desk Reference) can cause gynecomastia as a side effect. The risks are generally very low for male breast enlargement from these medications, but breasts in men can be a cause for embarrassment. Some medications may be important for your other medical problems.

Adalat
Aldactazide
Aldactone
Aldoclor
Al***e
Aldoril
Anadrol
Androderm
* Android
Atromid-S
Avonex
Axid
Baycol
Betaseron
Calan
Captopril
* Casodex
Catapres
Celexa
Cipro
Clinoril
Clorpres
Combipres
Compazine (No incidence data in labeling)
Covera-HS
Duraclon
Effexor
Elavil (No incidence data in labeling)
Etrafon
* Eulexin
Flexeril
Haldol
Humatrope
Humegon
Indocin
Intron A
Isoptin SR
Kadian
Lanoxicaps
Lanoxin
Lescol
Lexxel
Limbitrol (No incidence data in labeling)
Lotrel
Loxitane
Loxitane
Lupron
Matulane
Megace
Methotrexate
Mevacor
Midamor
Moban
Moduretic
Motrin
Myleran
Navane (No incidence data in labeling)
Neoral
* Nilandron
Nizoral
Norpace
Norpramin (No incidence data in labeling)
Norvasc
Novarel
Nutropin
Orudis
Oruvail
Oxandrin
Pepcid
Pergonal
Plendil
Pravachol
Pregnyl
Prevacid
PREVPAC
Prilosec
Procardia
Profasi (No incidence data in labeling)
Propulsid
Protropin
Prozac
Reglan
Repronex
Requip
Rifamate (No incidence data in labeling)
Rifater
Risperdal
Sandimmune
Sandostatin
SangCya
Serentil
Seroquel
* Serostim
Sinequan (No incidence data in labeling)
Sporanox
Stelazine (No incidence data in labeling)
Sular
Surmontil
Sustiva
* Tagamet
Tarka
* Testoderm
Testred (No incidence data in labeling)
Thalomid
Thioridazine Hydrochlorid
Thiothixene
Thorazine
Tiazac
Trecator-SC (No incidence data in labeling)
Triavil (No incidence data in labeling)
Tricor
Trilafon
Vascor
Vaseretic
Vasotec
Verelan
Vivactil (No incidence data in labeling)
Wellbutrin
Winstrol (No incidence data in labeling)
Xanax
Zantac
Zocor
* Zoladex
Zoloft
Zyban
Herbals
Digitalis Purpurea (FOXGLOVE)




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Dht & gyno
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Dht & gyno - 01-17-2013, 03:25 PM

In addition to elevated IGF-1, lowered DHT levels resulting from endogenous testosterone suppression may contribute to gyno from non aromatizing steroids. Gyno is a reported side effect from finasteride use. Some have attributed this to elevated estrogen levels due to the fact that there is more testosterone to be aromatized, since less test is being converted to DHT. Other researches think that DHT has a direct antiestrogenic effect on breast tissue.

Studies have shown that DHT can actually block estrogen from binding to the estrogen receptor in mammary tissue (1). DHT also is an aromatase inhibitor (2). Even more interesting is the fact that transdermal DHT cream has been used successfully to treat gyno (3).

It may be that the estrogen/DHT ratio is more important to the development of gyno than the estrogen/testosterone ratio.




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Progestins & GYNO
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Progestins & GYNO - 01-17-2013, 03:26 PM

Before you decide that blocking progesterone is the solution to gyno, consider a few things. There is not one case of progesterone induced gyno in the medical literature EXCEPT in those cases where strong synthetic progestins, like medroxyprogesterone, were administered. In these cases the gyno is due to suppression of LH and testosterone by the progestin, NOT by a direct effect on breast tissue. On a cycle your LH is already suppressed by the AAS anyway.

Breasts have two components: alveoli and ducts. The alveoli are what secrete milk; they drain into ducts. Gynecomastia is the result of ductal hyperplasia, not alveolar hyperplasia. Estrogen stimulates the ductal tissue, while progesterone stimulates the alveoli. Alveolar hyperplasia does not contribute to gyno. If you want to read more on breast development, I suggest visiting this site:

http://www.endotext.org/male/male14/male14.htm

In various tissues throughout the body, including cultured neoplastic breast tissue, progestins downregulate the estrogen receptor (1). Progesterone receptor blockers like RU-486 upregulate the estrogen receptor (1). This is consistent with the fact that RU-486 CAUSES gyno in patients in whom it is used to treat Cushing’s disease and meningiomas (2).

Progestins are also anti-estrogenic in that they induce the enzyme 17-hydroxysteroid dehydrogenase, which catalyzes the oxidation of estradiol to the less potent estrone. Progestins also induce estrogen sulfotransferase, the enzyme which catalyzes the sulfation and inactivation of estrogens.

So do progestins contribute to gyno, and if yes, how so? If you visit the link above you will see that progestins increase IGF-1 levels. As that article indicated, IGF-1 is essential to the the development of mammary tissue. This is also how it is believed that progestins in HRT or oral contraceptives contribute to breast cancer: by increasing IGF-1 levels. But as bodybuilders we are always trying to maximize IGF-1. Hence the futility of trying to lower IGF-1 by blocking progestins. The other anabolics we use will elevate (hopefully) IGF-1, while blocking the progesterone receptor will only increase the levels and activity of estrogen by the mechanisms outlined above.

Two drugs have shown the greatest efficacy in treating gyno: Nolvadex, and Raloxifene, another SERM. Nolvadex has the longest track record, but a recent trial with Raloxifene showed it to be superior to Nolvadex. With these drugs you attack the problem at its source: the estrogen receptor. You get the added benefit of lowering IGF-1. Not a good thing for making gains, but important for treating gyno.




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