The following pertains to DHT as well as Wintrol, Proviron, Masteron and Methyl-DHT, and probably some others like superdrol; and is a very basic explaination without details or references.
1. SHBG lowering -the high SHBG lowering causes the faster destruction of estrogen and without an increase in estrogen leads to a lowering by that effect.
(Winstrol has the advantage here with only needing 10mg to be as effective as 50mg of Proviron)
2. Causes a decreased estrogen-induced RNA transcription; basically estrogen cannot transcript its effects at the receptor due to a 'blockage' of sorts.
(Proviron and Masteron has the advantage here as being closer to purer DHT's)
3. DHT helps prevent estrogen-dependent augmentation of progesterone receptors, which is, as far as I can see the only way that progestins are capable of causing 'progesteronic' sides anyway.
(All seeming equal in this effect)
Of Note: This is not meant to imply that DHT compounds can replace AI's in an cycle, but merely to point out some of their effects in that regard; Doses and sensitivity would determine that.
1. SHBG lowering -the high SHBG lowering causes the faster destruction of estrogen and without an increase in estrogen leads to a lowering by that effect.
(Winstrol has the advantage here with only needing 10mg to be as effective as 50mg of Proviron)
2. Causes a decreased estrogen-induced RNA transcription; basically estrogen cannot transcript its effects at the receptor due to a 'blockage' of sorts.
(Proviron and Masteron has the advantage here as being closer to purer DHT's)
3. DHT helps prevent estrogen-dependent augmentation of progesterone receptors, which is, as far as I can see the only way that progestins are capable of causing 'progesteronic' sides anyway.
(All seeming equal in this effect)
Of Note: This is not meant to imply that DHT compounds can replace AI's in an cycle, but merely to point out some of their effects in that regard; Doses and sensitivity would determine that.
Last edited:
